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Dakota Peacock

Co-op/Honours Student Dakota Peacock




  • Tel:250-519-5716

  • Fax: 250-519-2040



Research Interest:


Tumour development relies in part on a transformed cell’s ability to evade detection by the immune system. Therefore, priming and directing the immune system against tumours holds promise as a means to combat cancer. CXCL10, the ligand for CXCR3, has been shown to play a role in effector T cell generation and trafficking in vivo. Currently, I am engineering vesicular stomatitis virus (VSV) and vaccinia virus (VV) to be used as an intravenous delivery vessel for the expression of CXCL10 at the cancer site. This work holds the potential to increase the presence of CD8+ T cells at the tumour site and to improve the survival rate of cancer patients.